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有关设置华工“海内外特出青年学者论坛”情状

2018年中华夏族民共和国矿业高校海外青年学者“越崎论坛”

广阔师生: 华南理工业余大学学学“海内外优异青少年学者论坛”旨在面向举世约请拥有差别学术背景的青少年才俊,围绕国际科学前沿、火热商量世界以及行当行业的能力难题等进行研讨和调换。希望藉此平台,相互启发、开垦视线,巩固国际调换与搭档,推动两岸联合提升。现将教院分论坛有关安插布告如下:一、论坛时间二〇一八年三月14日14:00-17:00二、论坛地点华南理哲高校高校城校区B2-513会议厅三、论坛章程

广泛师生:华工“海内外优良青年学者论坛”意在面向全世界诚邀青少年才俊,围绕国际科学战线、火爆切磋领域以及行当行当的技能难点等开展斟酌和交换。籍此平台,启迪思虑,开采视界,推动学术交换与搭档,这次论坛的具体布置如下:一、论坛时间二〇一八年二月8日早晨15:00-16:00二、论坛地方华工4号楼数学大学4318会议厅三、论坛议程1. 15:00-15:05高校领导致应接辞;2. 15:05-16:00报告人:沙敏硕士,澳大福冈(Australia)McCaw瑞大学,报告标题:代数数的乘性相关性(Multiplicative dependence of algebraic numbers)。招待广大师生参预!数学高校二零一八年三月12日内容摘要:代数数的乘性相关性是数论中的特出课题。大家称n个非零复数a1, ..., an是乘性相关的,假使存在不全为零的整数k1, ..., kn使得乘积a1k1... ankn=1。同理,称贰个n维向量是乘性相关的,假诺它的n个坐标是乘性相关的。报告将从深入分析、算术和几何角度进行。从深入分析角度,想念到稠密性难点。在实空间或复空间中,尽管乘性相关向量的勒贝格臆度为零,但表明它们在空中中是黑压压的,况且向量的坐标只需取自某些代数数域。然后经过考虑覆盖半径,那么些难点能够被更全面地钻探。从算术角度,思考到n维乘性相关向量的计数难题。举例,假若n维乘性相关向量的坐标是代数数,次数和惊人都有上界,那么对于那个向量的个数,给出了关于高度的渐近公式。从几何角度,思量到代数曲线上的乘性相关点。表明如若曲线定义在一个代数数域K上並且亏格大于零,那么曲线上独有有限个乘性相关点,其坐标来自于K的巨大Abe尓扩张。亏格为零的曲线也可能有左近结论。报告人简单介绍:沙敏,一九八四年7月诞生,大学生。2006年在华工得到博士学位,二〇一〇年在南开东军事和政院学得到大学生学位,二〇一二年在法兰西共和国也门萨这大学得到博士学位。二零一一年5月至2015年1十一月在澳大Cordova联邦(Commonwealth of Australia)新南Will士大学做大学生后。2015年7月到现在在澳大多特Mond联邦(Commonwealth of Australia)麦考瑞高校做校级大学生后。沙敏大学生长时间讨论数论及其使用。近些日子最首要讨论兴趣包罗代数数论,椭圆曲线,有限域理论,算术重力系统,线性递归系列,以及数论中的图论难题。在包蕴Transactions of the American Mathematical Society, International Mathematics Research Notices, Moscow Mathematical Journal, Journal of Combinatorial 西奥ry Series B等国际著名杂志上刊登杂文多篇。从二〇一二年起出任美利坚联邦合众国《数学争辨》的争辨员。于2011年获得欧洲联盟委员会的Alain Bensoussan Fellowship,并于二〇一五年赢得McCaw瑞大学的Macquarie University Research Fellowship。

布满师生:

财富高校分论坛

日期

附件:

华工“海内外卓越弱冠之年学者论坛”遇到与财富高校分论坛目的在于面向环球约请具备不一样学术背景的青少年才俊,围绕国际科学战线、火热探讨领域以及行业行当的本领难点等进行讨论和沟通。通过那个平台,相互启发、开辟视线,加强国际调换与搭档,推动两岸联合发展。现将情状与能源高校分论坛有关安排文告如下:

间:2018年6月6日-7日

时间

一、论坛时间

主办单位:能源与地学高校

须知或章程

2017年12月25日上午9:15-12:30

接待全校师生踊跃参预!

地点

二、论坛地方

率先会议室

10月23日

条件与财富大学B4-308会议场馆

地方:煤层气能源与成藏进度教育部第一实验室319会场

全天

三、论坛章程

(文昌校区西门外高校科学和技术园B座)

报到、入住;

时间

时间:2018年6月7日9点

高级学园城宗旨饭店

须知或议程

报告一:Particle-based modeling of pull-apart basin development

10月24日

9:15-9:30 开幕式

报告人:刘源,博士后, 德意志Frye贝格财政和经济科学和技术高校

14:00-17:00学术报告主持人:廉哲雄

高校总管致应接词

告诉摘录:

报告人:曾筑天(加拿大蒙Trey大学Cumming 管理高校博士后)标题:To See the Unseen: Intravital imaging reveals key immune defense mechanisms against bloodstream bacterial infection

9:30-12:30

1. A scale-independent modeling approach based on the Discrete Element Method has been built to investigate pull-apart basin development.

B2-513会议室

学术报告

2. The shape of a pull-apart basin is the consequence of both initial strike-slip geometry and its various evolution stages.

报告人:叶浩彬(罗德岛大学哲大学大学生后)标题:Management of Leukemia from Metabolic Perspectives

难题:大气污染物的遥感观测本事(Remote sensing observations of atmospheric aerosols and trace 瓦斯es)

3. Minimum displacements to form pull-apart basins, and minimum ages of initiation for pull-apart basins can be estimated.

应接广大师生参预! 哲大学二〇一八年四月十八日1.报告人:曾筑天(加拿大斯图加特高校Cumming 经济高校博士后)报告题目:To See the Unseen: Intravital imaging reveals key immune defense mechanisms against bloodstream bacterial infection内容摘要:Bloodstream bacterial infection is on the rise due to the wide spread use of indwelling intravenous catheters and immunosuppressive iatrogenic interventions. First-pass clearance of blood-borne bacteria is critical to control bloodstream infections and to prevent systemic dissemination. The liver has been well known as a blood-filtering organ capable of sequestering circulating bacteria via its vast pool of intravascular macrophages-Kupffer cells. However, the molecular mechanism underlying Kupffer cell mediated capture of circulating pathogens under shear conditions remains less understood. Taking advantage of high-speed real time intravital imaging, we visualized the dynamic process of bacterial capture by Kupffer cells, and revealed novel mechanisms utilized by Kupffer cells to catch bacteria. We observed a pattern recognition role for complement receptor-C逍客Ig in the capture of circulating Gram-positive bacteria from the bloodstream by directly binding to the Lipoteichoic Acid of Gram-positive bacteria, such as S. aureus. We also observed a sex-biased difference of Kupffer cells in capture circulating enteropathogenic E. coli . While complement opsonization was indispensable for the capture of EPEC in male mice; however, a faster, complement-independent process involving abundant preexisting antibodies to EPEC was detected in female mice. These antibodies were elicited predominantly in female mice at puberty in response to estrogen regardless of microbiota-colonization conditions. Estrogen-driven antibodies were maternally transferrable to offspring and conferred protection during infancy. Thus, an estrogen-driven, innate antibody-mediated immunological strategy conferred protection to females and their offspring. 报告人简单介绍︰曾筑天硕士于2000年至二〇〇八年就读于中国科学技术大学生命科学大学,获得学士学位。随后在中国科学技术大学免疫性学研究所师从田志刚院士并于贰零壹陆年7月获得细胞生物学大学生学位。2016年九月到现在,曾筑天大学生在加拿大圣多明各大学师从国际名牌免疫学专家PaulKubes教师进行大学生后钻探。在大学生及大学生后研商时期,曾筑天博士在肝脏天然免疫性学及感染应答机制等种类化做出一层层具有可持续性和立异型的首要切磋,主要行使高分辨率活体显微成像本事实现了对肝脏局地免疫性细胞和血液中微生物动态互相成效的实时成像观测,揭穿了肝脏抗细菌感染免疫性的积极分子细胞机制,开掘了斩新的由雌激素诱导的原生态抗体新群众体育,并对慢性病毒感染状态下肝脏免疫性效果低下致使乙型病毒性肝性传播病魔毒不可能被有效消除注解了机制。其讨论成果以率先作者身份公布在了Nature Immunology, Cell Host&Microbe, Journal of Experimental Medicine, Journal of Immunology等高素质免疫性学期刊上。曾筑天博士是免疫性学领域的优良青少年学者,多年来致力于肝脏病魔免疫性学调查研商及潜在免疫医治方法的开销,在肝脏免疫性这一最主要领域具备安如磐石的研商基础,具备明显合理的前程设计以及巨大的发展潜在的能量。曾筑天大学生明白卓殊的应用探究才能,能对活体小动物肝脏,肾脏,脾脏等多少个脏器免疫细胞进行直接实时动态的显微成像检查评定及剖判,是近日国际上为数十分的少的能对上述七个脏器进行活体动态成像商量的专家之一。 2.报告人:叶浩彬(内布拉斯加大学经济高校硕士后)报告标题:Management of Leukemia from Metabolic Perspectives内容摘要:Obese leukemia patients have a poorer prognosis compared to normal weight patients, suggesting that obesity-associated conditions protect leukemia cells/leukemia stem cells from chemotherapy. Further, obese population have a higher risk for leukemia, indicating that obesity promotes disease development and progression. However, the biological mechanisms underlying these phenomena remain unknown. In today’s presentation, the author introduces two studies that reveal the mechanisms for the phenomena mentioned above. The first study has demonstrated that adipose tissue functions as a sanctuary for LSCs. Briefly, LSCs are found to be enriched in adipose tissue. Tranome comparisons show that compared to hematopoietic tissue-resident LSCs, adipose-resident LSCs display a pro-inflammatory gene signature, which leads to an inflamed state in adipose tissue, and consequently an increased lipolysis rate as evidenced by elevated serum fatty acids level. Lipolysis-derived fatty acids are utilized by two distinct pathways: 1) fatty acids-induced inflammation pathway and 2) fatty acid oxidation pathway. Interestingly, a LSC subpopulation that has a high-level expression of the fatty acid transporter CD36 (CD36+ LSCs) displays a significantly higher FAO rate compared to CD36- LSCs and is strikingly enriched in adipose tissue. Further, CD36+ LSCs are more chemo-resistant compared to CD36- LSCs partially due to CD36-mediated FAO. More importantly, a CD36+ LSC subpopulation is also observed in primary human leukemia patient samples. Human CD36+ LSCs display a higher FAO rate and are chemo-resistant compared to CD36- LSCs. Collectively, this study shows that the interplay between leukemia cells and adipose tissue creates a unique microenvironment that supports the metabolic demands and survival of a distinct LSC population.The second study demonstrates that leukemic disease causes aberrancies in multiple tissues including adipose tissue, pancreas, gut and gut microbiota to subvert the systemic glucose metabolism to support growth of leukemia cells. Briefly, leukemia mice are found to have characteristics of both type 2 and type 1 diabetes: insulin resistance and insulin defect, conditions that inhibit glucose utilization in normal tissues. Mechanistically, leukemia induces a high-level production of IGFBP1 from adipose tissue, which results in insulin resistance. Further, leukemic disease impairs gut functions causing loss of gut-derived serotonin and dysbiosis. Serotonin loss results in inhibition of insulin secretion and dysbiosis leads to insulin resistance and less production of microbiota-derived short chain fatty acids such as butyrate and propionate. Supplementation of serotonin or SCFAs impedes leukemia progression. Importantly, combination of serotonin and SCFAs supplementations drastically reduce leukemic burden and prolong survival of leukemic mice by directly increasing the uptake and utilization of glucose in normal tissues. Together, these data demonstrate that restoration of normal glucose regulation may be a feasible strategy to suppress systemic growth of malignant cell types. Taking together, these two studies suggest that interventions by targeting either intracellular metabolism of LSCs or systemic metabolism are effective means for disease management.报告人简单介绍︰叶浩彬,博士,男,壹玖捌玖年诞生,结束学业于罗切斯特大学经济学院(University of Rochester Medical Center),现为亚利桑那高校管理大学(University of Colorado Medical Campus)博士后,主要开展白血病病理和白血病干细胞代谢及其微意况商量。已在Cancer Cell、 Cell Stem Cell、blood和Journal of Biological Chemistry等杂志上刊出杂谈及摘要8篇。担当Cancers、International Journal of Molecular Sciences、Molecules和Vaccines等国际学术期刊审阅稿件人。

报告人:陈嘉乐(德意志联邦共和国宇宙航行核心德文 Aerospace Center

个人简单介绍:

附件:

问题:基于高分辨质谱工夫的指向油砂工业废水的条件化学商讨

刘源,二零一八年三月大学生毕业于德意志Frye贝格理工科高校(TU Bergakademie Freiberg),大学生散文德国评分别得到得“magna cum laude”(非凡),现继续博士后商量。专门的学业是构造地质学,研讨方向为地质进度的数值模拟,大学生阶段师从国际盛名岩石力学和数值模拟专家HeinzKonietzky教师,学习用离散元数值模拟方法消除地质结构进程中的断裂扩展难题,属学科交叉研讨。第三回将离散元(DEM)方法运用到大标准的地质结构演化模拟中,建立了拉分盆地的断裂和盆地演变进度的数值模型,通过对模型尺度和断裂参数的剖判探讨,第三回基于离散元方法(颗粒流PFC)模拟了拉分盆地摇身一变演化进度中的断裂扩张难题,揭发了菱形拉分盆地的来源于难题,给出了总计拉分盆地形成的小不点儿位移和微时辰间的主意,为拉分盆地的变异演化进度提供了新的商讨思路和章程,相关成果以第一作者兼通信小编辑发表布于结构地质学界超级期刊Tectonics(IF:3.784)上。

报告人:黄荣夫(加拿大阿尔伯塔大学,副商量员)

报告二:Understanding the mechanical properties of shale in nanoscale

题目:生物成因煤层气的商量及商业化

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